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C221H366N72O67S
Tesamorelin
Tesamorelin binds to GHRH receptors, stimulating pulsatile GH release which elevates IGF-1. The elevated GH/IGF-1 axis promotes lipolysis in visceral adipocytes. An added trans-3-hexenoic acid moiety stabilizes the molecule and extends its half-life compared to native GHRH.
FDA-Approved GHRH Analogue for Visceral Fat
C14H23CuN6O4
GHK-Cu
GHK-Cu stimulates fibroblast proliferation and enhances the synthesis of collagen, glycosaminoglycans, and proteoglycans. It activates TGF-β signaling and modulates over 4,000 human genes involved in tissue remodeling, inflammation, and DNA repair. Its copper chelation properties contribute to antioxidant activity.
Copper Peptide — The Skin and Tissue Restorer
C14H22N4O9
Epitalon
Epitalon activates telomerase (hTERT), the enzyme responsible for adding telomeric repeats to chromosome ends, counteracting the shortening that occurs with each cell division. It also regulates the expression of genes involved in cell cycle control, apoptosis, and oxidative stress response. Interaction with the pineal gland normalizes melatonin and cortisol rhythms.
The Telomere Peptide — Resets the Biological Clock
C92H152N26O26S
MOTS-c
MOTS-c activates AMPK (AMP-activated protein kinase) and translocates to the nucleus under stress where it regulates nuclear gene expression. It inhibits the folate cycle and de novo purine biosynthesis, channeling resources to AMPK-mediated metabolic adaptation. It also modulates insulin receptor substrate signaling.
The Mitochondrial-Encoded Peptide for Metabolic Youth
C62H98N16O22
BPC-157
BPC-157 is thought to interact with the nitric oxide (NO) system and upregulate the expression of growth factors such as VEGF, EGF, and FGF. It appears to modulate the FAK-paxillin pathway, promoting cell migration and wound closure. Additionally, research indicates interactions with the dopaminergic and serotonergic systems.
The Body Protection Compound
C221H352N56O67
Retatrutide
Retatrutide is a 39-amino-acid peptide engineered from a GIP backbone with balanced agonist activity at three receptors. GLP-1 receptor activation suppresses appetite and improves glycemic control; GIP receptor activation enhances insulin sensitivity and fat metabolism; glucagon receptor activation increases hepatic energy expenditure, lipolysis, and fatty acid oxidation. The glucagon component is designed to drive additional fat loss without hyperglycemia because GLP-1 and GIP maintain glucose-dependent insulin secretion.
The Triple Hormone Receptor Agonist (GLP-1/GIP/Glucagon)
Research compound
Semax + Selank
Semax enhances monoaminergic system function and BDNF. Selank provides anxiolytic action through GABAergic and serotonergic systems.
Combined cognitive and anxiolytic peptide stack
C10H11N2
5-Amino-1MQ
NNMT methylates nicotinamide to produce 1-methylnicotinamide (1-MNA), consuming SAMe and depleting cellular NAD+ pools. This shifts adipocytes toward fat storage. 5-Amino-1MQ competitively inhibits NNMT, preserving intracellular NAD+ and SAMe levels. Elevated NAD+ activates SIRT1/PGC-1α signaling, increasing mitochondrial biogenesis and fatty acid oxidation. The compound also promotes expression of uncoupling proteins in white adipose tissue, shifting it toward a brown fat-like energy-expending phenotype.
NNMT Inhibitor — Small Molecule for Fat Oxidation
Research compound
Gutathione
Glutathione functions as the primary intracellular antioxidant, neutralizing reactive oxygen species through its thiol group.
Master antioxidant peptide for cellular protection
Research compound
HGH
HGH acts through binding to growth hormone receptors, activating the JAK2-STAT5 signaling pathway, stimulating hepatic IGF-1 production.
Human Growth Hormone (Somatropin) for growth and metabolic support
Research compound
AICAR
AICAR is taken up by cells and converted to ZMP (AICAR monophosphate), which mimics AMP and allosterically activates AMPK. This triggers downstream metabolic reprogramming: increased fatty acid oxidation, GLUT4 translocation for glucose uptake, mitochondrial biogenesis via PGC-1α, and inhibition of anabolic processes. The net effect mimics the metabolic state of endurance exercise.
The Exercise-Mimetic AMPK Activator
C46H56N12O6
GHRP-6
GHRP-6 binds the ghrelin receptor (GHS-R1a) on pituitary somatotrophs and simultaneously suppresses somatostatin tone, producing a strong GH secretory pulse. Its orexigenic effect arises from hypothalamic NPY and AgRP neuron activation, distinguishing it from GHRP-2 by a more pronounced appetite signal.
Growth Hormone Releasing Peptide-6
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